Anti-Slo2.1/Slick K+ Channel Antibody (N11/33)


Primary Antibody
Mouse, Rat
N11/33
IHC, WB
Mouse
Purified
Kcnt2
140 kDa

Go to Full Product Details


Anti-Slo2.1/Slick K+ Channel Antibody
SKU: 75-055

Volume: 100 µL
Price:
Sale price$308


Bulk Order Anti-Slo2.1/Slick K+ Channel Antibody

Product Specific References for Applications and Species

Electron Microscopy: Mouse
PMID Dilution Publication
289437561:500Tomasello, D.L., et al. 2017. Slick (Kcnt2) Sodium-Activated Potassium Channels Limit Peptidergic Nociceptor Excitability and Hyperalgesia. Journal of Experimental Neuroscience, .
Immunocytochemistry: Rat
PMID Dilution Publication
289437561:500Tomasello, D.L., et al. 2017. Slick (Kcnt2) Sodium-Activated Potassium Channels Limit Peptidergic Nociceptor Excitability and Hyperalgesia. Journal of Experimental Neuroscience, .
286602461:50Li, P., et al. 2017. GABA-B Controls Persistent Na+ Current and Coupled Na+-Activated K+ Current. eNeuro, .
270915441:50Bansal, V., et al. 2016. Na(+) -Activated K(+) Channels in Rat Supraoptic Neurones. Journal of Neuroendocrinology, .
Immunohistochemistry: Mouse
PMID Dilution Publication
289437561:250Tomasello, D.L., et al. 2017. Slick (Kcnt2) Sodium-Activated Potassium Channels Limit Peptidergic Nociceptor Excitability and Hyperalgesia. Journal of Experimental Neuroscience, .
291242161:200Rizzi, S., et al. 2015. Identification of potential novel interaction partners of the sodium-activated potassium channels Slick and Slack in mouse brain. Biochemistry and Biophysics Reports, 291-298.
Immunohistochemistry: Rat
PMID Dilution Publication
234668071:1000Cervantes, B., et al. 2013. Identity, expression and functional role of the sodium-activated potassium current in vestibular ganglion afferent neurons.. Neuroscience, 163-175.
194038311.5ug/mlChen, H., et al. 2009. The N-terminal domain of Slack determines the formation and trafficking of Slick/Slack heteromeric sodium-activated potassium channels. Journal of Neuroscience, 5654-5665.
Immunoprecipitation: Mouse
PMID Dilution Publication
2912421640ugRizzi, S., et al. 2015. Identification of potential novel interaction partners of the sodium-activated potassium channels Slick and Slack in mouse brain. Biochemistry and Biophysics Reports, 291-298.
26559620not listedMartinez-Espinosa, P.L., et al. 2015. Knockout of Slo2.2 enhances itch, abolishes KNa current, and increases action potential firing frequency in DRG neurons.. Elife, .
Western Blot: Mouse
PMID Dilution Publication
353030561:500Flauaus, C., et al. 2022. Slick Potassium Channels Control Pain and Itch in Distinct Populations of Sensory and Spinal Neurons in Mice. Anesthesiology, 802-822.
289437561:1000Tomasello, D.L., et al. 2017. Slick (Kcnt2) Sodium-Activated Potassium Channels Limit Peptidergic Nociceptor Excitability and Hyperalgesia. Journal of Experimental Neuroscience, .
26845140not listedWojtovich, A.P., et al. 2016. Cardiac Slo2.1 Is Required for Volatile Anesthetic Stimulation of K+ Transport and Anesthetic Preconditioning.. Anesthesiology, 1065-1076.
265879661:3000Rizzi, S., et al. 2016. Differential distribution of the sodium-activated potassium channels slick and slack in mouse brain.. Journal of Comparative Neurology, 2093-2116.
291242161:1000Rizzi, S., et al. 2015. Identification of potential novel interaction partners of the sodium-activated potassium channels Slick and Slack in mouse brain. Biochemistry and Biophysics Reports, 291-298.
26559620not listedMartinez-Espinosa, P.L., et al. 2015. Knockout of Slo2.2 enhances itch, abolishes KNa current, and increases action potential firing frequency in DRG neurons.. Elife, .
22145034not listedWojtovich, A.P., et al. 2011. SLO-2 is cytoprotective and contributes to mitochondrial potassium transport.. PLoS One, e28287.
Western Blot: Rat
PMID Dilution Publication
270915441:500Bansal, V., et al. 2016. Na(+) -Activated K(+) Channels in Rat Supraoptic Neurones. Journal of Neuroendocrinology, .
Western Blot: Xenopus
PMID Dilution Publication
29069600not listedGururaj, S., et al. 2017. A De Novo Mutation in the Sodium-Activated Potassium Channel KCNT2 Alters Ion Selectivity and Causes Epileptic Encephalopathy. Cell Reports, 926-933.

Related Products

Recently Viewed